HomeMy WebLinkAboutNCD003202603_20041224_Ward Transformer_FRBCERCLA RD_Public Health Assessment for Public Comment-OCR('~t(ATSDR
'<1> ~ AGe'.NCY FOR TOXIC SUBSTANCES
1::r;,1, AND DISEASE REGISTRY ""
Public, Health
Assessment
for -__ , -
SCANNED
OEC 1 0 2012
N.C. su~nd Section ~ ~e
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WARD TRANSFORMER ,...,,,.
RALEIGH, WAKE COUNTY-?NORTH CAROLINA V \\.. , EPA FACILITY(ID',N<?:D003202603 '" ''\,_, OCTOBER,28, 2004 ~❖-v·
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U.S. DEI?AR.1'M:E.N1' OIF lHIEAJL 1'H A.ND HUM:A.N SERVJICES
l?UJBJLIC lHIEAJL1'lHI §ERVJICE
Agency for Toxic Substances and Disease Registry
Comment Period Ends:
DECEMBER 27, 2004
THE ATSDR PUBLIC HEALTH ASSESSMENT: A NOTE OF EXPLANATION
This Puhlic Health Asscssmcnt-Puhlic Commcnl ·Release was prcpan.:d hy ATSDR pursuant to the Comprehensive
Environmental Response. Compensation. and Liahility Act (CERCLA or Snpcrfund) section 104 (i)(6) (42 U.S.C. 9604 (i)(6).
and in accordance with our implci11cming regulations (42 C.F.R. Part 90). In preparing this document, ATSDR has collected
relevant health data, environmental data, and community health concerns from !he Environmcnrnl Protection Agency (EPA),
st,ilc and local health and c.:nvimnmental agencies, the community, and potl.!1Hially rl.!sponsihlc panics, where appropriate.
This document represent::.; the agcncy1s best efforts, hascd on currently availahk information. to fullill the statutory critcrin set
ou! in CERCLA section i04 (i)(6) within a limited tim1: frame. To the cxtcnl possihlc, it presents an assessment of potential
risks 10 human health. Actions authorized hy CERCLA section !04 (i)( 11 ). or otherwise authorized hy CERCLA. may be
undertaken to prevent or mitigace human exposure or risks to human health. In addilion, ATS DR will utilize 1his document rn
delcrminc if follow-up health ac1ions are appropriate at 1his time,
This document has previously hccn provided to EPA and the affected state in an initial release, as n:quin:d hy CERCLA
st..:clion 104 (i)(6)(H) for their information and review. Where necessary. it has hccn revised in n:sponsc to comments or
additional rclcvanl information provided by ilu:m to ATSDR. · This: rcviScd· document-has now hecn released for a 30~day
puolic comment period, Suhsequcnt to the puhlic comment pcdotj,, Atsb1fwill address aH puhlic comments and revise or
append the document ,is appropriate. The puhlic health assessmerit will then he reissued:· This will conclude the public health
assessment process for this siw, unless additional information is o_htuinl:-d by ATSDR which. in lhc agency's opinion, indicates
a need to revise or append the conclusions previously issued, !
-,-::i. :-: ;
' Agency for Toxic Suhswnccs and Disease Registry .. , ........ ." ...... .Julie L. GcrbCrding, M.D .. M.P.H., Administrator
Henry Falk. M.D .. M.P.H., Assistant Administrator
Division of Health Asscssmcnl Jnd Consultation ... , ..... ,,....... , , , .William Cihulas, Jr., Ph.D., Dir:cctor
Sharon Williams-Fleetwood. Ph.D., Deputy Director
Community Involvement Branch ...... Germano E. Pereira. M.P.A., Chief
Exposure Investigations and Consultation Branch ...................... Susan W. Metcalf. M.D., M.S.P.H., Acting Chief
Federal Facilities Assessment Branch ...... . . , Sandra G. Isaacs, Chief
Program Evalumion, Records, and Information . . ...... Ma.x M. Howie, Jr., M.S., Chief
Supcrfund Site Asscssmcnl Branch .......... , . ........... Richard E. Gillig. M.C.P., Chief
Use of trade names is for identification only and docs not constitute endorsement by the Puhlic Health Service or the U.S.
Department of Health and Human Services.
Please address comments regarding this report to:
Agency for Toxic Suhstanccs and Disease Registry
Division of Health Assessment and Consul1ation
Attn: Chief, Program Evaluation, Records. and Information Scrvic<.:s Branch, E-60
1600 Clifton Road, N.E., Atlanta, Georgia 30333
You May Contact ATSDR TOLL FREE al
l-888-42ATSDR or
Visit our Home Page at http://www.atsdr.cd<:.go v
Ward Transformer
II
Public Comment Release
PUBLIC HEALTH ASSESSMENT
WARD TRANSFORMER
RALEIGH, WAKE COUNTY, NORTH CAROLINA
EPA FACILITY ID: NCD003202603
Prepared by:
Snperfnnd Site Assessment Branch
Division of Health Assessment and Consultation
Agency for Toxic Substances and Disease Registry
FORE\VORD
The Agency for Toxic Substances and Disease Registry, A TSDR, was established by Congress in 1980
under tbe Comprehensive Environmental Response, Compensation, and Liability Act, also kno1.vn as the
Superfund law. This law set up a fund to identify and clean up our country's hazardous waste sites. The
Environmental Protection Agency, EPA, and the individual states regulate the investigation and clean up
of the sites.
Since 1986, ATSDR has been required by law to conduct a public health assessment at each of the sites on
the EPA National Priorities List. The aim of these evaluations is to find out if people are being exposed to
hazardous substances and, if so, whether that exposure is harmful and should be stopped or reduced.
(The legal definition ofa health assessment is included on the inside front cover.) If appropriate,
ATS DR also conducts public health assessments when petitioned by concerned individuals. Public health
assessments are carried out by environmental and health scientists from ATSDR and from the states with
which A TSDR has cooperative agreements. The public health assessment program allows the scientists
flexibility in the format or strncture of their response to the public health issues at hazardous waste sites.
For example, a public health assessment could be one document or it could be a compilation of several
health consultations the structure may vary from site to site. Nevertheless, the public health assessment
process is not considered complete until the public health issues al the site are addressed.
Exposure: As the first step in the evaluation, A TSDR scientists review environmental data to see how
much contamination is at a site, where it is, and how people might come into contact with it. Generally,
ATS DR does not collect its own environmental sampling data but reviews information provided by EPA,
other government agencies, businesses, and the public. When there is not enough environmental
infonnation available, the report will indicate what further sampling data is needed.
Health Effects: lflhe review of the environmental data shows that people have or could come into
contact with hazardous substances, ATS DR scientists evaluate whether or not these contacts may result in
harmful effects. ATSDR recognizes that children, because of their play activities and their growing
bodies, may be more vulnerable to these effects. As a policy, unless data are available to suggest
otherwise, A TSDR considers children to be more sensitive and vulnerable to hazardous substances. Thus,
the health impact to the children is considered first when evaluating the health threat to a community. The
health impacts to other high risk groups within the community (such as the elderly, chronically ill, and
people engaging in high risk practices) also receive special attention during the evaluation.
ATS DR uses existing scientific information, which can include the results of medical, toxicologic and
epidemiologic studies and the data collected in disease registries, to determine the health effects that may
result from exposures. The science of environmental health is still developing, and sometimes scientific
information on the health effects of certain substances is not available. \Vhen this is so, the report will
suggest what further public health actions are needed.
Conclusions: The report presents conclusions about the public health threat, if any, posed by a site. Wben
health threats have been determined for high risk groups (such as children, elderly, chronically ill, and
people engaging in high risk practices), they will be summarized in the conclusion section of the report.
Ways to stop or reduce exposure will then be recommended in the public health action plan.
Public Health Assessment -Public Comment Release Ward Transformer NPL Site
Calculation of Risk of Carcinogenic Effects ............................................................................. 26
Appendix R Exposure Pathways for Ward Transformer Site ...................................................... 28
Appendix C. ATSDR Glossary of Environmental Health Tenns ................................................ 29
Public Health Assessment Public Comment Release Ward Transformer NPL Site
Acronyms and Abbreviations
AL
ATSDR
coc
CREG
CSF
CV
EMEG
EPA
ES!
FDA
HOD
IARC
IUPAC
LOAEL
MCL
mg/kg/day
' mglm"
MRL
NCDENR
NIOSH
NOAEL
Nl'DES
NPL
OSHA
PCB
PHA
ppb
ppm
ppl
R3RBC
R9PRG
RID
RI
RMEG
SSL
TCDD
TEF
TEQ
~tg/L
WHO
U.S. EPA Action Level
Agency for Toxic Substances and Disease Registry
Contaminant of Concern
Cancer Risk Evaluation Guide
Cancer Slope Factor
Comparison Value
Environmental Media Evaluation Guide
U.S. Environmental Protection Agency
Expanded Site Inspection
Food and Drug Administration
Health Outcome Data
International Agency for Research on Cancer
International Union of Pure and Applied Chemistry
Lowest Observed Adverse Effect Level
Maximum Contaminant Level
milligram per kilogram per day
milligram per cubic meter
Minimal Risk Level
North Carolina Department of Environment and Natural Resources
National Institute for Occupational Safety and Health
No Observed Adverse Effect Level
National Pollutant Discharge Elimination System
National Priorities List
Occupational Safety and Health Administration
Polychlorinated biphenyl
Public Health Assessment
part per billion
part per million
part per trillion
U.S. EPA Region 3 Risk-based Concentration
U.S. EPA Region 9 Preliminary Remediation Goal
Reference Dose
Remedial Investigation
Reference Media Evaluation Guide
U.S. EPA Soil Screening Level
Tetrachlorodibenzodioxin
Toxicity Equivalency Factor
Toxicity Equivalency Quotient
microgram per liter
World Health Organization
Public Health Assessment -Public Comment Release Ward Transformer NPL Site
Summary
The Ward Transformer site is an operating transformer recycling facility located on Mount
Herman Road in Raleigh, North Carolina. The facility is in a mostly industrial area close to U.S.
70, l-540, and the Raleigh-Durham International Airport. The facility has been in operation since
about l 964. Before l 977, when the use of PCBs in transformer oil was discontinued, PCBs
contaminated soils on the site and surface water and stream sediments downstream of the site.
On the basis of the information available about the Ward Transformer site, ATSDR concluded
the following;
I. Edible portions offish from areas downstream of the site have PCBs at levels high
enough to increase the theoretical risk of cancer and adverse noncancer health effects for
people who eat these fish regularly. The State o[North Carolina has placed an advisory
against eating carp or catfish from Lake Crabtree or any species of fish from Brier Creek,
Brier Creek Reservoir, Little Brier Creek downstream of Brier Creek Parkway, and the
tributary leading from the Ward Transformer site. The State ofNorth Carolina also
advises that consumption of fish species other than carp or catfish from Lake Crabtree be
limited to no more than I meal per month.
2. Exposure of site workers to PCBs in soil could contribute to an increased theoretical risk
of developing cancer.
3. Exposure lo PCBs in sediment and surface waler is not a significant contributor to overall
theoretical cancer risk. However, sediments may contribute to PCB contamination in the
aquatic food chain. As stated above, consumption of contaminated fish could increase the
risk of cancer and adverse noncancer health effects.
4. The groundwater beneath the site is not being used for drinking water and therefore is not
of public health concern at this time. However, limited data suggest that the groundwater
might be impacted by site contaminants. Not enough information exists to detem1ine
whether health effects could be possible if the groundwater was used for drinking.
Because exposure to PCBs in fish and/or soil could increase the risk for cancer or adverse
noncancer health effects if exposure is not reduced and/or prevented, ATSDR classifies the Ward
Transformer site as a public health hazard.
ATSDR has made the following recommendations about the site:
I. ATSDR recommends that the public follow fish advisories placed by the State of North
Carolina.
2. ATSDR recommends action be taken to minimize exposure of employees at Ward
Transformer to PCBs in soil,
3. ATSDR recommends that action be taken to reduce the contribution of contaminants in
sediment to the food chain.
4. If groundwater beneath the site is used for drinking water in the future, it should be fully
characterized lo detem1ine its impact on public health.
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Public Health Assessment -Public Comment Release Ward Transfom1er NPL Site
Purpose and Health Issues
The Ward Transformer site was proposed for the National Priorities List (NPL) on September 5,
2002 and listed on the NPL on April 30, 2003. Congress requires A TSDR to conduct public
health activities on all sites proposed for or listed on the NPL. In this public health assessment
(PHA), A TSDR evaluates the public health significance of the Ward Transforn1er site. ATSDR
reviewed environmental data, potential exposure pathways, and community health concerns lo
determine whether adverse health effects are possible. In addition, this PHA recommends actions
to prevent, reduce, or further identify the possibility for site-related adverse health effects.
Background
Site Description
The Ward Transfornier site is an operating transformer recycling facility located on Mount
Hennan Road in Raleigh, Wake County, North Carolina. The facility is in a mostly industrial
area close to U.S. 70, 1-540, and the Raleigh-Durham International Airport. The facility has been
in operation since about 1964. Before 1977, when the use ofpolychlorinated biphcnyls (PCBs) in
transformer oil was discontinued, PCBs contaminated soils on the site and surface water and
stream sediments downstream of the site. The Ward Transformer company constructed a
stormwater retention pond in 1972 and a water treatment plant in 1979. These treatment
processes have operated consistently within permit requirements.
Demographics
Figure I shows demographic infomiation for the area surrounding the site. Approximately l 04
people, including about 12 children under age 6, live within a 1-mile radius of the site. The
population around the site is about 85% Caucasian, I 0% African American, and 4% Asian.
Land and Natural Resource Use
The Ward Transformer site covers about 11 acres in a sparsely populated area outside of Raleigh,
North Carolina. The site is surrounded on three sides by other industrial properties and highways,
and abuts land belonging to the Raleigh-Durham International Airport. The airport land is
restricted, and the site and most of the surrounding industrial properties are fenced. Mount
Herman Road dead-ends into a major highway (U.S. 70) a few hundred feet north of the site
before continuing on the north site of U.S. 70.
The facility was constructed on previously undeveloped land in 1964 and has reconditioned
· transformers since that time. The main yard of the operating facility contains the reconditioning
facility, offices, and hundreds of used transformers stored for possible resale. Some of the
transformers have been on site for more than IO years. The northern portion of the yard is now
vacant but previously was leased to a forest products company and may have been used to store
or recondition transfonners before that. The yard is almost completely paved (some areas are
aged and cracked) and is surrounded by an 8-foot high chain-link fence topped with barbed wire.
2
Figure I. Site Location and Demographic Information, Ward Transformer, Raleigh, NC
,d
I Ward Transformer
Raleigh, North Carolina
EPA Facility ID NCD003202603
Bas!½ Map Soorce: 1995 TIGER/Line Filss
Population Density
Adults 65 Years and Older
JVA120302
0 0.5 1 -= Seate in Miles
0 0.5 1 -= Scale in Miles
3
~NTRO MAP
Site location
7
Wake County. North Carolina
Demographic Statistics
Within One Mile of Site•
Total Population 104
White alone 88
Black alone 10
Am. Indian and Alaska Native alone o
Asian alone 4
Native Hawaiian and
Other Pacific Islander alone o
Some other race alone o
Two or More races 1
Hispanic or Latino 2
Children Aged 6 and Younger 12
Adults Aged 65 and Older 5
Females Aged 15 • 44 25
Total Housing Units 39
Demographics Statistics Source: 2000 US Census
"Calrutated using an area-proportion spatial ana~is technique
Children 6 Years and Younger
Females Aged 15 -44
Scuoe: 2000 us. een..,,
D US Census Block
i Zero Pop. ,1.ation 1 • 9 Children
10 -20 Children
> 20 Children
0 0.5 1 -= Scale in Miles
0 0.5 1 -= Scale ln Miles
Public Health Assessment -Public Comment Release Ward Transforn1er NPL Site
Also in the main yard is a bumoff oven (similar to an incinerator), which was installed in 1973
and burned transfonner oil in the past.
Before I 972, surface water runoff flowed overland to intennittent streams west and south of the
facility. In 1972, two unlined lagoons were built in a fenced area immediately west of the main
yard of the site for stormwater retention. A National Pollutant Discharge Elimination System
(1'<1'DES) pem1itted stormwater treatment plant and curbs to direct all stormwater runoff from the
facility to the lagoons were installed in 1979. This treatment plant removes PCBs from water in
the storm water lagoon before releasing it to an unnamed stream that joins Little Brier Creek to
the west and eventually flows into Lake Crabtree about 2 miles west and south of the site. The
site is in compliance with its NPDES pern1it requirements.
PCBs have been measured in sediments and surface water in and around the creek ri1ore than a
mile downstream of the site. The nearest downstream residences are approximately 1 mile
downstream of the site; the residences appear to be at a higher elevation than the creek.
Groundwater is thought to flow toward the creeks. The only drinking water use of groundwater
identified near the site was at a business located immediately north of the site, Employees of this
business used an onsite drinking water well until 1995, The facility has been connected to the
municipal water supply since April 1995.
Discussion
Data Used
The data used in this evaluation came from the following sources:
• Sampling of soil, sediment, and incinerator ash as part of the 1998 Expanded Site Inspection
(ES!) perfom1ed by the North Carolina Department of Environment and Natural Resources
(NCDENR) [J]. The ESJ Report also presented results of soil and sediment sampling from
past investigations, including the 1995 Site Inspection by the North Carolina Superfund
Section, a 1993 Removal Investigation by the U.S. Environmental Protection Agency(EPA),
and sampling in the late 1970s by the EPA.
• Sampling of soil, sediment, surface water, groundwater, and fish as part of the 2003 remedial
investigation by EPA. EPA' s contractor provided the data through the Remedial Tnvestigation
and Risk Assessment report revised in May 2004 [4],
The conclusions reached in this document are based on the data available at the time and might
be modified on the basis of results of additional samples collected in the future.
ATS DR staff (Jill Dyken, Shan-Ching Tsai, Ruby Palmer, and Benjamin Moore) visited the site
in March 2003. A walk-through to observe the main features of the site was conducted with EPA
representatives (Luis Flores and Diane Barrett) and EPA contractors. The team observed the
following:
4
Public Health Assessment -Public Comment Release Ward Transformer NPL Site
• The fence around the facility was approximately 8 feet high, topped with barbed wire, and in
good repair. Three sides around the lagoons was fenced with a tall fence in good repair, but
the west side had only a very short fence (about 2 feet high) that could easily be stepped over.
All fences had locks that could be accessed only by Ward employees.
• Most of the main yard was paved, but the pavement was aged and cracking in some areas.
Hundreds of used transformers were densely packed in the yard.
• The fonner Horizon Forest Products building was vacant.
• About a dozen employees were reconditioning transformers inside the warehouse building on
the Ward site.
• A number of 55-gallon drums were stored on pallets in the main yard near the burnoff oven.
• The burnoff oven was located in the main yard, between the warehouse building and the water
treatment facility.
Also, ATS DR staff drove through the area surrounding the site to better understand the
relationship between the site and the people living and working nearby. A TS DR staff observed
the following:
• Surrounding industrial facilities had many cars in the parking lots, indicating daily worker
populations in the area.
• One house was located about 300 feet northeast of the facility. This house had formerly been
repmied as occupied, but A TSDR could not determine whether it was currently occupied.
• Other than the one house, the areas immediately surrounding the facility were industrial
properties, land belonging to the Raleigh-Durham airport and marked prominently with "No
Trespassing" signs, or vacant land buffering the two major highways crossing near the Ward
facility.
• Commercial and residential development is occurring on the other side of 1-540,
approximately I mile west ( downstream) of the site.
Evaluation Process
The process by which ATSDR evaluates the possible health impact of contaminants is
summarized here and described in more detail in Appendix A. The first step involves screening
data for contaminants of concern (COCs). ATSDR uses comparison values (CVs) to determine
which chemicals to examine more closely. CVs are concentrations of chemicals in the
environment (air, water, or soil) below which adverse human health effects are not likely.
Exceeding a CV does not mean that health effects will occur, just that more evaluation is needed.
ATSDR also considers sampling location, data quality and community health concerns in
determining which chemicals to evaluate further.
If a chemical contaminant is selected for further evaluation, the next step is to identify which
chemicals and exposure situations could be a health hazard. Exposure doses for children and
adults are calculated for COCs in site media (e.g., soil, groundwater, surface water, sediment,
fish or shellfish). Exposure doses are the estimated amounts of a contaminant to which people
come in contact under specified exposure situations. These exposure doses are compared with
appropriate health guidelines for that chemical. Health guideline values are considered safe
5
Public Health Assessment -Public Comment Release Ward Transfom1er NPL Site
doses; that is, adverse health effects are unlikely below this level. If the exposure dose for a
chemical is greater than the health guideline, then the exposure dose is compared with known
health effect levels identified in ATSDR's toxicological profiles and other current reference
sources. lf the COC is a carcinogen, the theoretical cancer risk is also estimated. These
comparisons provide the basis for determining whether the exposure is a health hazard.
Because the Ward Transformer business involved recycling used transformers since the l 960s,
the main contaminants of concern at the site are PCBs, which were used in transfonners before
1977. Data were screened for all contaminants of concern, but an overview of PCBs is given here
because they are likely to be the most important contaminants of concern at this site and because
completed pathways for exposure were identified.
Polycblorinated Biphcnyls
Overview
PCBs are a group of synthetic organic chemicals that exhibit insulating and flame retardant
properties. They were first manufactured and used in the late l 920s. PCBs were used widely as
coolants and lubricants in transfonners and other electrical equipment and as process chemicals
in the manufacture of common products including plastics, fluorescent lighting fixtures, and
pesticides. PCBs are also relatively inert and take a long time to break down. In 1977, evidence
that PCBs build up in the environment and may cause harmful effects led to a halt in the
manufacture of PCBs in the United States. However, PCBs are still found throughout the world
because they break down so slowly and may be transported long distances in the environment
People can be exposed to PCBs in air, water, soil, or sediments. Most exposure to PCBs,
however, probably results from eating contaminated fish or meat Because PCBs break down so
slowly, tissue levels of PCBs tend to increase up the food chain as predators eat smaller species.
In this way, the fish and meat people eat can be contaminated with higher levels of PCBs than
those existing in the air, water, soil, or sediments.
Chemical Structure
The chemical structure of PCBs determines their toxicity. The base of every PCB is the biphenyl
molecule, which consists of two benzene rings joined together (Figure 2), Hydrogen atoms are
attached to each of the numbered corners representing carbon atoms in the diagram. To form a
PCB, 2-10 chlorine atoms substitute for these hydrogen molecules [5].
Figure 2. Bi phenyl Chemical Structure (from 161)
3· z· 2 l
5· 6"
Chlorine atoms can substitute for hydrogen at any of the 10 numbered carbon atoms shown. The
209 possible combinations that can be formed are known as PCB congeners. One numbering
6
Public Health Assessment -Public Comment Release Ward Transformer NPL Site
system for naming PCBs is derived from the carbon atom numbers shown in Figure 2. Positions
2, 2', 6, and 6' are called ortho positions, positions 3, 3 ', 5, and 5' are called meta positions, and
positions 4 and 4' are called para positions. The benzene rings can rotate around the bond
connecting them; when the rings are in the same plane, they are referred to as planar or coplanar
PCBs. Planar PCBs have a structure similar to the chlorinated dioxins and furans and generally
are more toxic than nonplanar PCBs. When the relatively large chlorine atoms are attached in
opposing ortho positions, the molecule cannot lie flat; therefore, only non-or/ho or mono-ortho
PCBs can assume the planar configuration.
An alternative way to name PCB congeners is using a numbering system based on International
Union of Pure and Applied Chemistry (IUPAC) rules for substituent characterization in
biphenyls (5]. Using this system, congeners PCB-I through PCB-209 are ordered, with
increasing numbers increasing in degree and complexity of substitution, according to standard
IUPAC rules.
The major U.S. producer of PCBs until 1977 marketed mixtures of PCBs under the trade name
Aroclor. The Aroclors are identified by a 4-digit numbering code in which the final two digits
indicate the approximate weight percentage of chlorine in the mixture (e.g., Aroclor 1260
contains about 60% chlorine). Each Aroclor is a mixture of various PCB congeners.
Health Effects Caused by PCB Exposure
Exposures of workers to high levels of airborne PCBs-many times higher than are likely to be
present at Ward Transformer today-caused skin conditions, such as acne and rashes, and
changes in blood and urine that might indicate liver damage. In animal studies, long-term oral
exposure to PCBs caused various health effects, including liver damage, skin conditions,
impaired reproduction, and immunologic and behavioral changes. PCBs are not known to cause
serious birth defects. However, evidence exists that children exposed to PCBs in the womb
(through their mother's cons·umption of contaminated fish) had neurobehavioral and
developmental deficits (5,7]. In human and animal studies, PCBs are associated with certain
kinds of cancer, such as cancer of the liver and biliary tract (5]. EPA considers PCBs probable
human carcinogens. The evidence of PCBs' carcinogenicity in animal studies is sufficient and
current evidence in humans is inadequate but suggestive [8].
Certain PCB congeners may exhibit toxicity similar to the chlorinated dibenzodioxins and
dibenzofurans, structurally similar molecules. One of the most toxic and most studied of these
molecules, 2,3,7,8-tetrachlorodibenzodioxin (2,3,7,8-TCDD), can cause acne-like skin lesions,
rashes, liver damage, hormonal changes, and increase the theoretical risk of cancer [9]. Toxicity
of dioxin-like PCB congeners can be expressed as a fraction of the toxicity attributed to 2,3,7,8-
TCDD, or a toxicity equivalency factor (TEF) [9, 10, 11]. Table 1 shows the TEF weighting
factors used for the PCB congeners measured at the Ward Transformer site. Individual PCB
congeners are multiplied by their respective TEF and summed, along with comparably weighted
dioxins and furans, to obtain the toxicity equivalency quotient (TEQ). The TEQ can be compared
directly with 2,3,7,8-TCDD toxicity information. This public health assessment evaluated PCBs
7
Public Health Assessment Public Comment Release Ward Transformer NPL Site
individually and (when congener data were available) using TEQs to detennine !he specific
dioxin-like toxicity.
Table!. TEF \Vehahtin2 Factors or P B f C C on2eners at the ar ran W d T sformer Site
Congener Number TEF Weighting Factor
PCB Congener #105 0.0001
I PCB Congener # 1 14 0.0005
PCB Congener #118 0.0001
PCB Congener #123 0.0001
PCB Congener #126 i 0.1
; PCB Congener # l 56 0.0005
PCB Congener #157 0.0005 '
PCB Congener # l 67 0.00001
PCB Congener# 169 0.01
PCB Congener #189 0.0001
; PCB Congener #77 0.0001
PCB Congener #81 0.0001
Current Stamlanls, Reg11latio11s, a11d Recomme11datio11s
EPA has set a regulatory maximum contaminant level (MCL) for PCBs in drinking water as 0.5
parts of PCBs per billion parts of water (ppb). For protection of human health from eating fish or
shellfish from surface waters, EPA recommends that the level of PCBs in surface waters be no
greater than 0.17 parts per trillion (ppl).
The State of North Carolina placed fish advisories for surface waters leading from the Ward
Transformer site on the basis of the level of PCBs in fish from Lake Crabtree, Brier Creek, Little
Brier Creek, Brier Creek Reservoir, and the tributary leading from the Ward Transformer site
[12,13]. No other fish advisories based on PCBs were localed in North Carolina [14]. The fish
consumption advisories recommend that no fish be eaten from Brier Creek (downstream of Brier
Creek Reservoir), Brier Creek Reservoir, Little Brier Creek (downstream of Brier Creek
Parkway), or the ullllamed tributary to Little Brier Creek. ln addition, the advisories recommend
that no carp or catfish from Lake Crabtree be eaten, and all other fish from Lake Crabtree be
eaten at no more than one meal a month.
The Food and Dmg Administration (FDA) has set residue limits (i.e., tolerances) for PCBs in
various foods to protect consumers from harmful health effects. FDA limits include 0.2 parts per
million (ppm) in infant and junior foods, 0.3 ppm in eggs, 1.5 ppm in milk and other dairy
products, 2 ppm in edible portions of fish and shellfish, and 3 ppm in poultry and red meat [ 15].
The Occupational Safety and Health Administration (OSHA) requires that workers not be
exposed by inhalation over a period of 8 hours for 5 days per week to more than l milligram per
cubic meter of air (mg/m3) for PCBs containing 42% chlorine or to 0.5 mgim3 for PCBs
containing 54% chlorine. The National Institute for Occupational Safety and Health (NIOSH)
recommends that workers not breathe air containing PCBs at levels higher than 0.001 mg/m3 for
a I 0-hour workday, 40-hour workweek (16].
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Public Health Assessment -Public Comment Release Ward Transformer NPL Site
The National Toxicology Program and the International Agency for Research on Cancer (]ARC)
have classified PCBs as reasonably anticipated to be carcinogens and as probably carcinogenic to
humans, respectively. EPA has classified PCBs as a probable human carcinogen. EPA has
calculated an upper bound oral slope factor of2 per milligram per kilogram per day
(2 [mg/kg/dayr1) to be used for such exposures as food chain exposure, sediment or soil
ingestion, presence of dioxin-like congeners, and early-life exposures. EPA also calculated less
conservative oral slope factors for use in other situations (for example, when toxic PCB
congeners make up less than½% of the mixture or for more water-soluble congeners) [8].
Complete Exposure Pathways and Contaminants of Concern
Fish Pathway
People might eat fish caught from waters downstream from the site. Because site contaminants
(particularly PCBs) build up over time in fish tissue, people who eat the fish might be exposed to
contaminants by eating the fish.
Composite samples of fish were collected in May 2003 during the remedial investigation. Fish
were sampled from the unnamed tributary to Little Brier Creek and the Brier Creek Reservoir.
Whole body samples of fish from the unnamed tributary were collected, and both whole body
and file! (with skin) samples were collected from the Brier Creek Reservoir. ATSDR used all the
May 2003 data to screen for contaminants of concern, and further evaluation was performed
using only the fish file! data, since it is assumed recreational fishers in the area do not eat the
whole fish.
To evaluate the potential for health effects from eating the fish, ATSDR assumed the 95 th
percentile fish ingestion rate for freshwater recreational anglers of25 grams per day for adults
[I 7]. Small children weighing 22 pounds were estimated to consume 12.5 grams of fish per day,
on average. These consumption rates correspond to about one fish meal per week, where adults
eat 6 ounces of fish per meal and small children eat 3 ounces of fish. These assumptions are
considered conservative for recreational consumption of fish from Brier Creek Reservoir. The
reservoir is posted with "Do Not Eat Fish" signs, but ATSDR received anecdotal reports of
people fishing there in the past. Also, fishers are unlikely to fish exclusively on the Brier Creek
Reservoir, as assumed in this evaluation.
Table 2. Contaminants of Potential Concern in Comoosite Fish File! Samoles
Maximum Estimated Health Health Guideline Excess Cancer
Contaminant Composite File! Dose for Child, Guideline, Source (defined Risk, if Concentration, mg/kg/day mg/kg/day in Appendix A) applicable mg/kg
Total mercury I.I 0.001 0.0003 Chronic MRL for Not Applicable orgamc mercury
Aroclor 1260 2.6 0.003 0.00002 Chronic MRL for 7 in 10,000 PCBs
'Based on 25-year exposure duration at adult dose of0.00093 mg/kg/day.
Source: r 41
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Public Health Assessment -Public Comment Release Ward Transformer NPL Site
As shown in Table 2, two contaminants resulted in estimated exposure doses higher than health
guidelines. These contaminants will be evaluated in the following paragraphs.
Mercury
The form of mercury Iha! builds up in fish is methylmercury, an organic (carbon-containing)
form of mercury. High levels ofmethylmercury can result in brain and kidney damage and can be
especially harmful to developing children exposed either prenatally or after birth. The highest
level of mercury measured in fish fi!ets downstream of the Ward Transformer site is I. I mg/kg,
only slightly higher than FDA's action level of I mg/kg for commercial seafood. The estimated
child dose of0.001 mg/kg/day is about the same as the dose of0.0013 mg/kg/day that had no ·
adverse health effects in human epidemiologic studies; however, other studies have suggested
developmental effects may occur at similar doses [l 9]. Mercury is a widespread contaminant and
was not known to be used at the Ward Transformer company. Although a slightly increased risk
for health effects is possible from exposure to mercury through this pathway, Ward Transformer
is unlikely to have contributed significantly to this risk.
Aroclor /260
Aroclor 1260 consists of a mixture of PCBs. The estimated intake for children eating fish caught
recreationally is 0.003 mg/kg/day and for adults is 0.0009 mg/kg/day. The chronic-duration
minimal risk level derives from an animal study in which Rhesus monkeys fed as little as 0.005
mg/kg/day Aroclor 1254 for 23 months exhibited decreased antibody response and some mild
clinical manifestations of toxicity (eyelid and toe/fingernail changes) [5]. An intem1edia1e-
duration minimal risk level (0.00003 mg/kg/day) is derived from an animal study in which
monkeys fed as little as 0.0075 mg/kg/day for 20 weeks exhibited decrements in learning and
neurobehavioral performance. For both children and adults, estimated doses for recreational
consumption of fish are within an order of magnitude of the lowest observed adverse effect levels
observed in these studies. If children and adults ate enough fish, adverse health effects could
result. The most likely health effects would be mild immunologic or neurologic changes.
The excess cancer risk associated with eating filets from recreationally caught fish with the
average Aroclor l 260 concentration listed in Table 2 was estimated, assuming daily and
eontinual exposure over a 25-year period. The estimated theoretical excess cancer risk is about 7
in I 0,000; ATS DR considers this a \ow-to-moderate increased risk for cancer. The actual risk of
developing cancer depends on many factors, including actual consumption, actual contaminant
levels in the fish consumed, genetics, lifestyle, and other environmental factors.
Update of Aroc/or 1260 Analysis Based on November 2003 Fish Sampling
In November 2003, additional fish samples were collected further downstream from the May
2003 sampling locations. Fish were collected from the lower portion of Brier Creek Reservoir,
from Brier Creek downstream from Brier Creek Reservoir and from three different areas in Lake
Crabtree. Whole body samples were collected from Brier Creek, and both whole body and filet
samples were collected from Brier Creek Reservoir and from Lake Crabtree. Filets had skin left
on except for catfish filet samples which had the skin removed. The November fish samples were
10
Public Health Assessment -Public Comment Release Ward Transformer NPL Site
analyzed only for PCBs, PCB congeners, and dioxins/furans. A TSDR evaluated only the fish filet
results, since it is assumed recreational fishers in the area do not eat the whole fish. A summary
of all of the Aroclor 1260 results, showing location, species, and average file! concentration of
Aroclor 1260 for each species and location collected, is shown in Table 3.
T able 3. Fish 1let F' S ampling Result Summary, \Vard Transformer s itc, Raleigh, N C
Location Fish Species Average Composite Filet Aroclor
1260 Concentration in mo/ko
Brier Creek Reservoir Blueeill sunfish 0.5
LarP-emouth bass 1.6
Brown bullhead 1.7
Yellow bullhead 1.2
Lake Crabtree Blueeill sunfish 0.2
Larvemouth bass 0.2
Common cam 0.3
Channel catfish 1.2
Assuming the same recreational consumption rates as above, all the filet concentrations in Table
3 would result in increased risk of cancer and non cancer health effects. Because of the lower
PCB levels in Lake Crabtree sunfish and bass, limiting consumption of these fish to no more than
I meal a month would be expected to minimize the risk of adverse health effects.
Soil I11gestio11 Pathway
Employees working on the site or people trespassing on or near the site could come into contact
with contaminated soil. They could get particles of the soil on their skin, or they might
unintentionally eat or breathe in the particles. Soil from the site has been sampled and analyzed
for contaminants. Although people generally are exposed only to surface soil no more than 3
inches below ground surface, A TSDR used results from samples taken from 0-12 inches below
ground surface to estimate surface concentration, since that was the only depth range available.
This may overestimate or underestimate the actual concentration of contaminants to which
people are exposed at the site. Table 4 lists the contaminants that were detected at least once in
surface soil above the corresponding soil CV.
Table 4. Surface Soil Contaminants of Concern at Ward Transformer Site, Raleigh, NC
Contaminant Maximum concentration Comparison CV Source (defined
in soil-nnm Value (CV), nnm in Annendix Al
Arsenic 2 20 / 0.5 EMEG/CREG
Copper 6,300 2,900 R9PRG
Iron 30,000 23,000 R9PRG
DDT 33 30 I 2 RMEG/CREG
Aldrin 0.061 2 / 0.04 EMEG/CREG
Dieldrin 5.2 3 I 0.04 EMEG/CREG
Heptachlor Epoxide 0.21 0.7 / 0.08 RMEG/CREG
Aroclor 1260 1,700 0.4 CREG
Dioxin/Furan/PCB TEQ 0.06 0.00005 EMEG
~ource: [4]
Maximum of anv single PCB congener.
1 I
Public Health Assessment -Public Comment Release Ward Transformer Nl'L Site
For further screening, worst-case exposure doses for the contaminants listed in Table 4 were then
estimated for adult workers, assuming they were exposed to the maximum concentrations listed.
ln addition, although trespassing appears unlikely in the vicinity of the site, we estimated
exposure doses for 10-year-olds who might regularly trespass on the site and be exposed to the
maximum concentrations above. Details of the assumptions used in performing these
calculations can be found in Appendix A. The estimated exposure doses for children and adults
of arsenic, copper, iron, DDT, aldrin, dicldrin, and heptachlor epoxide were below noncancer
health guideline values and resulted in a less than l in I 0,000 excess cancer risk, and these doses
are therefore not expected to result in any adverse health effects.
For further evaluation of Aroclor 1260 and dioxins/furans/PCBs, the average concentrations in
surface soil were calculated because the long-term exposure would be to an average
concentration rather than the maximum. These contaminants were evaluated in two ways: 1)
Aroclor 1260 exposure was used to estimate risk using PCB toxicologic information, and 2) the
TEQ for dioxins, furans, and dioxin-like PCB congeners (weighted for toxicity and summed) was
used to assess risk using toxicologic infonnation for 2,3,7,8-TCDD. Aroclor 1260 surface soil
concentrations ranged from less than 0.036 ppm to 1,700 ppm in 67 samples collected at and in
the immediate vicinity of the site. The average Aroclor 1260 was 89 ppm. Eleven of the surface
soil samples also were analyzed for dioxins, furans, and specific PCB congeners. TEP-corrected\
concentrations in soils were summed to obtain the TEQ of total dioxin-like compounds. The
TEQs in surface soil samples ranged from 0.00005 ppm to 0.06 ppm, and the average was 0.01
ppm as 2,3,7,8-TCDD.
Exposure 10 Aroclor 1260 in Surface Soil
Because Aroclors are mixtures of both dioxin-like and non-dioxin-like PCB congeners, summing
the risk associated with both the Aroclor and with individual congeners included in the TEQ
could overestimate risk by accounting for the dioxin-like PCB congener risk both individually
and within the risk estimates for Aroclor. However, because the mass of dioxin-like PCB
congeners is only about 2% of that of Aroclor, correcting the Aroclor concentration by
subtracting out congener concentrations had a negligible effect on both the average concentration
of Aroclor and on the calculated risk. Therefore, ATSDR proceeded with the evaluation using the
uncorrected Aroclor value.
The average Aroclor 1260 concentration in surface soil is 89 ppm, which results in an average
estimated doses of0.00014 mg/kg/day and 0.00009 mg/kg/day for child trespassers and adult
workers, respectively. The chronic-duration minimal risk level (0.00002 mg/kg/day) is derived
from an animal study that found a lowest observed adverse effect level of0.005 mg/kg/day [5].
The child dose is more than 30 times smaller than this level, and the worker dose is more than 50
times smaller. In addition, the actual exposure to Aroclor in surface soil would probably be
smaller than estimated because access is restricted so that trespassing is highly unlikely, most of
the site is paved, and workers most frequently access areas of the site that are paved. ATSDR
considers adverse noncancer health effects from exposure to soil unlikely.
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Public Health Assessment -Public Comment Release Ward Transformer NPL Site
Aroclor 1260 is a probable human carcinogen. The contribution from soil exposure to theoretical
excess cancer risk for a 25-year exposure period to an adult is about 6.5 in 100,000. Although
this is a low cancer risk, it contributes to risk from multiple pathways, which will be addressed
later.
Exposure to Dioxin-Like PCBs in Surface Soil
The average TEQ for dioxin-like PCBs in surface soils, 0.01 ppm, corresponded to estimated
child and adult exposure doses of l .6x 10·3 and l .Ox 10·3 mg/kg/day, respectively. The minimal
risk level for 2,3,7,8-TCDD is based on a monkey study that showed altered social behavior at a
lowest observed effect level of l .2x I 0·7 mg/kg/day. The estimated doses arc an order of
magnitude lower than this level. Adverse noncancer health effects are not expected from
occasional trespassing or worker exposure to PCB congeners in soil.
The theoretical excess cancer risk associated with exposure to dioxin-like PCBs in soil for a 25-
year duration is about 5.5 in 10,000. ATSDR considers this a low-to-moderate increased risk for
cancer. The overall cancer risk from multiple exposure pathways will be addressed later.
Sediment Pathway
People who trespass on impacted creek beds downstream from _the site might unintentionally
ingest some of the sediments from the creek or get the sediments on their skin. Sediment CV s
were not available, so sediment CVs were set at 10 times the corresponding soil CV because
sediment was assumed to be contacted one tenth as much as soil particles, so the concentration of
contaminant could be 10 times as high for the same dose. As shown in Table 5, Aroclor 1260
was detected above the corresponding sediment CV.
Table 5. Sediment Contaminants of Concern at Ward Transformer Site, Raleieh, NC
Contaminant Maximum Comparison CV Source
concentration in Value (CV) fo~ (defined in
sediment, nnm sediment, nnm Annendix A)
Aroclor 1260 62 4 Soil CREG x I 0
• Sediment CV calculated as 10 times the soil CV because sediment ingestion was assumed to be one tenth
of the average soil ingestion.
Source: f 41
ATSDR calculated an exposure dose for trespassers who contact Aroclor 1260 in sediment
4 times a week throughout the year for 25 years. The excess risk for cancer associated with such
contact is not significantly elevated. However, the risk from exposure to Aroclor 1260
contributes to the cancer risk from multiple exposure pathways, which will be discussed later.
13
Public Health Assessment-Public Comment Release Ward Transformer l\'PL Site
Surface Water Pathway
No use of surface water downstream of the site for drinking water was identified, but people who
wade or swim in this water will get surface water on their skin and could ingest some of it.
Incidental ingestion of the surface water was assumed to be no more than one tenth the nonnal
drinking water ingestion. Therefore, surface water CVs were calculated as 10 times the drinking
water CV. Table 6 lists the contaminants that were detected at least once above the
eorresponding surface water CV.
T s f able 6 . ._ ur ace \Vater C ontaminants o fC V dT oncern at \ ar f rans ormcr 1te, a eJ:2..1,,. s· R I ' l NC
f\'1aximum concentration in Surface \Voter CV Source (defined in I Contaminant Comporison Value surface water, µg/L (CV), ne/1/ Ap1icndix A) I
Manganese 7300 5000 Drinking Waler I RcvlEG x 10
Aldrin 0.044 3/0.02 ! Drinking Water
EMEG / CREG x 10
Heptachlor Epoxide 0.055 I /0.04 Drinking Water
RMEG / CREG x 10
Aroclor 1260 ! 1.5 0.2 Drinking Water
I CREG x 10
• Surface water CV calculated as ten times the drinking water CV.
Source: r41
For further screening, worst-case exposure doses for the contaminants listed in Table 6 were then
estimated for trespassers who contact the maximum concentrations in surface water 4 times a
week throughout the year over many years. Details of the assumptions used to perform these
calculations can be found in Appendix A. The estimated ehild and adult exposure doses for
manganese, aldrin, and heptachlor epoxide were below cancer and noncancer health guideline
values and therefore not expected to result in any adverse health effects. In addition, the excess
cancer risk associated with exposure to Aroclor 1260 in surface water is not significantly
elevated. However, exposure to this contaminant contributes to the cumulative risk for cancer
from multiple exposure pathways, which will be discussed later.
Cumulative Exposures
People could be exposed to site contaminants through more than one of the pathways discussed
in this document. ln this section, we consider whether multiple exposure pathways might result
in an increased health risk over the pathway-specific analysis. Tahle 7 shows the relative
contribution to overall theoretical cancer risk of all the pathways for Amel or and PCB congeners.
As indicated, most of the risk is contributed by the fish consumption and soil pathways. The
sediment and surface water pathways have a negligible contribution to the overall risk. If a
person were exposed to PCBs through all four pathways, the theoretical increased risk for cancer
is about 2 in 1,000. ATSDR considers this a moderate-to-high increased risk for cancer. [t should
be noted that worst-case assumptions were used to obtain the theoretical cancer risk. The actual
risk of developing cancer, which is likely to be much smaller than the upper bound estimate
reported here, depends on many factors, including actual exposures, genetics, lifestyle, and other
environmental factors.
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Public Health Assessment -Public Comment Release Ward Transformer NPL Site
Table 7. Contribution of Pathways and Contaminants to Overall Theoretical Cancer Risk, \Vard
Pathwav
Fish
Soil
Sediment
Surface Water
Cumulative Risk
of Contaminant
Potential Exposure Pathways
Groundwater Pathway
Transformer Site, Ralei2"h, NC
Dioxin/Fu ran/PCB Cumulative Risk of
Aroclor 1260 TEO Pathwav
6.6x10"4 4.Sx 10·4 1.0x 10·3
6.Sx 10·5 5.Sx 10-4 6.2x 10·4
2.8x 10·7 4.7x 10·10 2.8x 10·7
7.0xlO·' -7.0x 10·7
7.3 x10·4 1.0x I 0·3 1.7x10"3
No use oflocal groundwater for drinking water was identified in the vicinity of the site. Three
wells near the site were previously used for drinking water. The well at Ward Transformer
reportedly had been tested yearly before the company was connected to the municipal water
supply in 1994; no contaminants were found above drinking water standards. Two other nearby
wells were sampled in August 1994: a residential well had no detections of contaminants, and a
well at a former auto shop had detectable petroleum constituents, at levels below drinking water
standards. Therefore, because no evidence exists of actual exposure to harmful levels of
contaminants in these wells, the groundwater pathway is considered incomplete.
ATSDR evaluated monitoring well data collected from the shallow groundwater during the RI.
Data were available from 2 of the 5 monitoring wells installed on and around the Ward
Transformer site. Some contaminants (including PCBs, chlorinated benzenes, pesticides, and
some metals) were detected at levels above drinking water CVs (data not shown). ATSDR
recommends that the groundwater beneath the site not be used for drinking unless the water is
fully characterized to determine its public health impact.
Air Pathway
No measurements of air emissions from the facility are available. Although the bumoff oven on
site has never been permitted to bum used transformer oil containing PCBs as fuel, it is known
that some burning of PCB oil did occur during at least one year, and possibly more, in the past.
This could have released dioxins from the incomplete burning of PCBs.
In the 1997 ESI, sampling of ash from the bumoff oven and in the soil directly underneath the
oven showed high levels of metals, dioxins/furans, and Aroclor 1260. Ash from the bumoff oven
is currently drummed and shipped offsite for disposal, so this pathway is incomplete. Because the
pathway is currently incomplete and because no information exists to allow an evaluation of
potential past exposures, this pathway will not be considered further.
15
'
Public Health Assessment -Public Comment Relea~e Ward Transfonner l\'PL Site
Children's Health Considerations
ATSDR recognizes that infants and children might be more vulnerable than adults to exposures
in communities with contaminated air, water, soil, or food. This potential vulnerability results
from the following factors: 1) children are more likely to play outdoors and bring food into
contaminated areas; 2) children are shorter and therefore more likely to contact dust and soil; 3)
children's small size results in higher doses of chemical exposure per kg of body weight; and 4)
developing body systems can sustain permanent damage if toxic exposures occur during critical
growth stages. Because children depend completely on adults for risk identification and
management decisions, ATSDR is committed to evaluating their special interests at the site.
Because of the limited access to the site and the surrounding areas, ATSDR considers small
children unlikely to be directly exposed to site contaminants. Small children might be affected by
previous exposure in the womb from their mothers' consumption of fish. Small children also
might eat contaminated fish. Older children who trespass on nearby property might be exposed to
contaminants in the surface water and sediments of streams downstream of the site. Refer to the
appropriate section for discussion of the possible health effects for these exposure pathways.
Health Outcome Data
Health outcome data (HOD) can give a more thorough evaluation of the public health
implications ofa given exposure. HOD can include mortality information (e.g., the number of
people dying from a certain disease) or morbidity infonnation (e.g., the number of people in an
area getting a certain disease or illness). The review is most effective when (I) a completed
human exposure pathway exists, (2) contaminant levels are high enough to result in measurable
health effects, (3) enough people are affected for the health effect to be measured, and (4) a
database is available to identify disease rates for populations of concern,
A review of health outcome data was not performed for this site. Although completed exposure
pathways exist al this site, the potentially exposed population is too small to allow statistical
differences in the rates of occurrence of relatively uncommon diseases to be measured.
Community Health Concerns
ATSDR staff attended a public meeting at the Morrisville Commerce Building in Morrisville,
North Carolina, on March 13, 2003, EPA organized the meeting to discuss the Ward Transfonner
site. Approximately IO community members and 15 local, state, and federal officials attended the
meeting. A TSDR discussed the PHA process and asked community members to share their
health concerns related to contaminants at the site. Following are concerns expressed by
members of the audience at the meeting; ATSDR responses were developed a~er the meeting as
part of producing this public health assessment:
Concern: Are workers of the businesses around Ward Transformer exposed to hazardous levels
of contaminants from the site?
16
Public Health Assessment -Public Comment Release Ward Transformer NPL Site
Response: Currently available data suggests that soil at surrounding businesses has not been
impacted by the site. Assuming that workers at other businesses do not frequently contact the soil
at the Ward Transformer site, no adverse health effects would be expected. EPA is still in the
process of completing the remedial investigation for the site and determining the extent of
contamination. They will present the findings at a public meeting.
Concern: ls fish from Lake Crabtree safe to eat?
Response: In May 2004, the State of North Carolina issued an advisory against eating carp and
catfish from Lake Crabtree. They also advised people to limit consumption of all other fish from
Lake Crabtree to 110 more than one meal per month. In addition to the advisory about Lake
Crabtree, North Carolina has also issued advisories against eating any fish from Brier Creek
downstream of Brier Creek Reservoir, Brier Creek Reservoir, Little Brier Creek downstream of
Brier Creek Parkway, and one unnamed tributary to Little Brier Creek.
Concern: Flooding could have spread contamination beyond the areas already measured.
Response: Although flooding might have dispersed contaminants from the site, such dispersal
would have diluted the contaminants, so that very high levels would be unlikely to be contacted.
Also, the contaminants would be difficult to measure accurately. ATSDR considers EPA 's
remedial investigation adequate to fully characterize the nature and extent of potentially
hazardous contamination from the Ward Transformer site.
Health Hazard Category
The levels of PCBs in edible portions of fish from areas downstream of the site are high enough
to increase the risk for cancer and adverse noncancer health effects for recreational levels of
consumption. In addition, exposure of workers to PCBs in soil could contribute to the potential
risk of developing cancer. Therefore, A TSDR classifies the Ward Transformer site as a public
health hazard because potential exposures to PCBs could result in adverse health effects if
exposure is not reduced or prevented.
Although the theoretical risk calculations suggest increased risk for adverse health effects for
workers and people eating fish from Brier Creek reservoir, it is important to note that these
calculations were based on worst-case exposure scenarios. The actual exposures are likely to be
much lower, so that the risk of adverse health effects occurring is low.
Conclusions
I. Edible portions of fish from areas downstream of the site have PCBs at levels high
enough to increase the theoretical risk of cancer and adverse noncancer health effects for
people who eat these fish regularly. The State of North Carolina has placed an advisory
against eating carp or catfish from Lake Crabtree or any species of fish from Brier Creek,
Brier Creek Reservoir, Little Brier Creek downstream of Brier Creek Parkway, and the
tributary leading from the Ward Transformer site. The State of North Carolina also
17
Public Health Assessment -Public Comment Release Ward Transformer NPL Site
advises that consumption of fish species other than carp or catfish from Lake Crabtree be
limited to no more than I meal per month.
2. Exposure of site workers to PCBs in soil could contribute lo an increased theoretical risk
of developing cancer.
3. Exposure to PCBs in sediment and surface water is not a significant contributor to overall
theoretical cancer risk. However, sediments may contribute to PCB contamination in the
aquatic food chain. As stated above, consumption of contaminated fish could increase the
risk of cancer and adverse noncancer health effects.
4. The groundwater beneath the site is not being used for drinking water and therefore is not
of public health concern at this time. However, limited data suggest that the groundwater
might be impacted by site contaminants. Not enough infonnation exists to determine
whether health effects could be possible if the groundwater was used for drinking.
Recommendations
1. ATSDR recommends that the public follow fish advisories placed by the State ofl\orth
Carolina.
2. ATSDR recommends action be taken to minimize exposure of employees at Ward
Transformer to PCBs in soil.
3. ATSDR recommends that action be taken to reduce the contribution of contaminants in
sediment to the food chain.
4. If groundwater beneath the site is used for drinking water in the future, it should be fully
characterized to determine its impact on public health.
Public Health Action Plan
The public health action plan for the Ward Transformer site describes actions that have been or
will be taken at the site by A TSDR and/or other government agencies. The purpose of the plan is
to ensure that this PHA not only identifies public health hazards at the site, but also outlines a
plan of action to prevent or minimize the potential for adverse human health effects from
exposure to site-related hazardous substances. ATSDR will follow up on this plan to ensure that
it is implemented.
Completed Actions
• ATSDR conducted a site visit to verify site conditions and to gather pertinent information and
data for the site.
• ATSDR attended a public meeting to inform the community about the public health
assessment process and to gather health concerns from the site community.
• The State of North Carolina issued an advisory against eating fish from Brier Creek Reservoir,
Little Brier Creek downstream from Brier Creek Parkway, and the unnamed tributary of Little
Brier Creek leading from Ward Transfonner.
• The State of North Carolina issued an advisory against eating fish from Brier Creek
downstream of Brier Creek Reservoir, catfish or carp from Lake Crabtree, and more than one
meal per month of other fish species from Lake Crabtree.
18
Public Health Assessment -Public Comment Release Ward Transformer NPL Site
Planned Actions
• ATSDR will review additional environmental sampling results for the site to evaluate any
changes in possible public health implications.
19
Public Health Assessment -Public Comment Release
Site Team
Authors of Report
Jill J. Dyken, Ph.D., P.E.
Environmental Health Scientist
Division of Health Assessment and Consultation
Shan-Ching Tsai, Ph.D.
Toxicologist
Division of Health Assessment and Consultation
Regional Repr<::sentative
Benjamin Moore
Regional Representative
ATSDR Region IV
Community Involvement
Ruby Palmer
Community [nvo!vement Specialist
Division of Health Assessment and Consultation
20
Ward Transfonner NPL Site
Public Health Assessment -Public Comment Release Ward Transformer !\'PL Site
References
I. North Carolina Department of Environment and Natural Resources, Expanded site
inspection for Ward Transformer site. Raleigh: North Carolina Department of
Environment and Natural Resources, Division of Waste Management; July 1998.
2. Weston Solutions, Inc. Ward Transformer site sampling and analysis plan and site
management plan. Prepared for the U.S. Environmental Protection Agency, Region 4.
West Chester (PA): March 2003.
3. U.S. Environmental Protection Agency. NPL site narrative for Ward Transformer. Atlanta
(GA): U.S. Environmental Protection Agency Region 4; 2002. URL:
http:i/wv,w.epa.govisuperfund/sitesl!ml/narl666.htm Accessed April 29, 2003.
4. Weston Solutions, Inc. Ward Transformer site revised remedial investigation and risk
assessment report. Prepared for the U.S. Environmental Protection Agency Region 4.
West Chester (PA): May 2004.
5. Agency for Toxic Substances and Disease Registry. Toxicological profile for
polychlorinated biphenyls (update). Atlanta: U.S. Department of Health and Human
Services; November 2000.
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program research findings. URL: http://www.atsdr.cdc.gov/grtlakes/research-
findings.htmJ Accessed March 17, 2004.
8. U.S. Environmental Protection Agency. Integrated risk information system (for
polychlorinated biphenyls). URL: http://www.epa.gov/i1islsubstl0294.htm Accessed
November 20, 2003.
9. Agency for Toxic Substances and Disease Registry. Toxicological profile for chlorinated
dibenzo-p-dioxins (update). Atlanta: U.S. Department of Health and Human Services;
December 1998,
I 0. Van den Berg M, Birnbaum L, Bosveld AT, Brunstrom B, Cook P, Feeley M, et al. Toxic
equivalency factors (TEFs) for PCBs, PCDDs, PCDFs for humans and wildlife. Environ
Health Perspect 1998; 106(12):775-792.
I I. U.S. Enviromnental Protection Agency, Columbia River Basin fish contaminant survey,
1996-1998. Seattle: U.S. Environmental Protection Agency, Region 10; July 2002.
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12. State of North Carolina Department of Health and Human Services. Press release entitled
"Fish consumption advisory issued for lower reaches of Little Brier Creek and Brier
Creek Reservoir areas; officials say that little fishing occurs on contaminated area."
Raleigh: North Carolina Department of Health and Human Services; December 2003.
13. State of North Carolina Department of Health and Human Services. Press release entitled
"Warning issued against eating certain fish from Lake Crabtree, all fish from Brier
Creek." Raleigh: North Carolina Department of Health and Human Services; May 2004
14. North Carolina Division of Public Health. Current fish consumption advice and
advisories in North Carolina. Raleigh: North Carolina Division of Public Health,
Epidemiology Section. URL: http://www.schs.state.nc.us/epi/fish/current.html Accessed
November 24, 2003.
15. Code of Federal Regulations. Tolerances for polychlorinated biphenyls (PCBs). 21 CFR,
Sect. 109.30 (2003).
16. National Institute for Occupational Safety and Health. Online NIOSH pocket guide to
chemical hazards. URL: http://www.cdc.gov/niosh/npg/npgdOOOO.html Accessed
November 20, 2003.
17. U.S. Environmental Protection Agency. Exposure factors handbook. Washington: U.S.
Environmental Protection Agency, Office of Research and Development; 1999. Rpt. No.:
EP N600/C-99/00 I.
18. Institute of Medicine. Dietary reference intakes for vitamin A, vitamin K, arsenic, boron,
chromium, copper, iodine, iron, manganese, molybdenum, nickel, silicon, vanadium, and
zinc. 2001.
19. Agency for Toxic Substances and Disease Registry. Toxicological profile for mercury
(update). Atlanta: U.S. Department of Health and Human Services; 1999 March.
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Appendix A. Explanation of Evaluation Process
Screening Process
In evaluating these data, ATSDR used comparison values (CVs) to determine which chemicals to
examine more closely. CVs are the health-based thresholds for contaminant concentrations found
in a specific media (air, soil, or water). They are used in the selection of contaminants for further
evaluation. A CV incorporates assumptions about daily exposure to a chemical and the standard
amount of air, water, and soil that someone might inhale or ingest each day.
A CV represents a concentration below which no known or anticipated adverse human health
effects are expected to occur. Different CVs are developed for cancer and noncancer health
effects. Noncancer levels are based on valid toxicologic studies for a chemical, with appropriate
safety factors included, and the assumption that small children (22 pounds or less) and adults are
exposed every day. Cancer levels are based on a one-in-one-million excess cancer risk for an
adult eating contaminated soil or drinking contaminated water every day for 70 years. For
chemicals for which both cancer and noncancer levels exist, ATSDR uses the lower of the levels
to be protective of human health. However, exceeding a CV does not mean that health effects
will occur; it merely means that more evaluation is needed.
The CV s used in the evaluation in this document are listed below:
Environmental Media Evaluation Guides (EMEGs) are estimated contaminant concentrations in
a media at which noncarcinogenic health effects are unlikely. EMEGs are derived from the
Agency for Toxic Substances and Disease Registry's (A TSDR) minimal risk level (MRL).
Cancer Risk Evaluation Guides (CREGs) are estimated contaminant concentrations that would
be expected to cause no more than one additional excess cancer in one million persons exposed
over a lifetime. CREGs are calculated from the U.S. Environmental Protection Agency's (EPA)
cancer slope factors (CSFs).
Reference Media Evaluation Guides (RMEGs) are estimated contaminant concentrations in a
media at levels at which noncarcinogcnic health effects are unlikely. RMEGs are derived from
EPA's reference dose (RID).
Region 9 Preliminary Remediation Goals (R9 PR Gs) are the estimated contaminant
concentrations in a media at which carcinogenic or noncarcinogenic health effects are unlikely.
The PRGs used in this PHA were derived by use of provisional reference doses or CSFs
calculated by EPA's Region 9 toxicologists and were last updated in October 2002.
Region 3 Risk-based Concentrations (R3 RBCs) are the estimated contaminant concentrations in
a media at which carcinogenic or noncarcinogenic health effects are unlikely. The RBCs used in
this PHA were derived by use of provisional reference doses or CSFs calculated by EPA's
Region 3 toxicologists and were last updated in October 2003.
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EPA Action Levels (ALs) are the estimated contaminant concentrations in water at which
additional evaluation is needed to determine whether action is required to eliminate or reduce
exposure. Action levels can be based on mathematical models.
EPA Soil Screening Levels (SSLs) are estimated contaminant concentrations in soil at which
additional evaluation is needed to determine if action is required to eliminate or reduce exposure.
World Health Organization guidelines (WHO) are guidelines published by the World Health
Organization for drinking water quality.
Some CV s may be based on different durations of exposure. Acute duration is defined as
exposure lasting 14 days or less. Intermediate duration exposure lasts between 15 and 364 days,
and chronic exposures last one year or more. Comparison values based on chronic exposure
studies are used whenever available. If an intermediate or acute comparison value is used, it is
denoted with a small i or a before the CV (e.g., iEMEG refers to the intermediate duration
EMEG).
Determination of Exposure Pathways
ATSDR identifies human exposure pathways by examining environmental and human
components that might lead to contact with COCs. A pathway analysis considers five principal
elements: a source of contamination, transport through an environmental medium, a point of
exposure, a route of human exposure, and an exposed population. Completed exposure pathways
are those for which the five elements are evident, and indicate that exposure to a contaminant has
occurred in the past, is now occurring, or will occur in the future. Potential exposure pathways
are those for which exposure seems possible, but one or more of the elements is not clearly
defined. Potential pathways indicate that exposure to a contaminant could have occurred in the
past, could be occurring now, or could occur in the future. It should be noted that the
identification of an exposure pathway does not imply that health effects will occur. Exposures
might be, or might not be, substantive. Therefore, even if exposure has occurred, is now
occurring, or is likely to occur in the future, human health effects might not result.
A TSDR reviewed site history, information on site activities, and the available sampling data. On
the basis of this review, ATSDR identified numerous exposure pathways that warranted
consideration.
Evaluation of Public Health Implications
The next step is to take those contaminants present at levels above the CVs and further identify
which chemicals and exposure situations are likely to be a health hazard. Child and adult
exposure doses are calculated for the site-specific exposure scenario, using our assumptions of
who goes on the site and how often they contact the site contaminants. The exposure dose is the
amount of a contaminant that gets into a person's body. Following is a brief explanation of how
we calculated the estimated exposure doses for the site.
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Fislt ingestion
Exposure doses for ingestion of fish downstream of the site were calculated using the average
concentration measured in fish tissue samples, in mg/kg or ppm, multiplied by the 95 th percentile
ingestion rate for recreational fishers of 25 grams per day (g/day). An ingestion rate of 12.5 g/day
was assumed for children. The calculated value was also multiplied by a conversion factor of
0.001 kilograms per gram. The multiplication product was divided by the average weight for an
adult (70 kg or 154 pounds) or a one-year-old child (10 kg or 22 pounds) to obtain the exposure
dose in mg/kg/day.
Soil lllgestion
Exposure doses for ingestion of contaminants present in soil were calculated by use of the
maximum concentration measured in soil, in milligrams per kilogram (mg/kg), or parts per
million (ppm). This maximum concentration is then multiplied by the soil ingestion rate for
adults (100 mg/day) or children (200 mg/day). For worker exposures, the multiplication product
was divided by the average weight for an adult, 70 kg (154 pounds). The resulting dose was then
multiplied by a factor of 5/7, because the exposure was assumed to occur five days a week
throughout the year. For exposure of child trespassers, the body weight used was 36 kg (80
pounds) and the exposure factor was 2/7 (2 days a week throughout the year).
Surface Water Ingestion
Exposure doses for surface water ingestion were calculated by use of the maximum concentration
for a surface water contaminant, in milligrams per liter (mg/L). This maximum concentration was
then multiplied by an incidental surface water ingestion rate of0.2 L/day for adults or 0.1 [Jday
for children. These ingestion rates are l /10th of the EPA default drinking water rates. The
multiplication product was divided by the average weight for an adult (70 kg) or for a I 0-year-old
child (36 kg). The resulting dose was then multiplied by a factor of 4/7, because the exposure
was assumed to occur four times a week throughout the year.
Sediment fllgestio11
Exposure doses for ingestion of contaminants from the sediment were calculated by use of the
maximum concentration measured in the sediment, in mg/kg or ppm, multiplied by I/10th of the
default soil ingeslion rate-IO mg/day for adults or 20 mg/day for children. The multiplication
product was divided by the average weight for an adult (70 kg) or a IO-year-old child (36 kg).
The resulting dose was then multiplied by a factor of 4/7, because the exposure was assumed to
occur four times a week throughout the year. The ealculated value was also multiplied by a
conversion faetor of0.001 kilograms per gram.
Noncancer Health Effects
The calculated exposure doses are then compared to an appropriate health guideline for that
chemical. Health guideline values are considered safe doses; that is, health effects are unlikely
below this level. The health guideline value is based on valid toxicological studies for a
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chemical, with appropriate safety factors built-in to account for human variation, animal-to-
human differences, and/or the use of the lowest adverse effect level. For noncancer health effects,
the following health guideline values are used.
Minimal Risk Level (MRLs) -Developed by ATSDR
An MRL is an estimate of daily human exposure -by a specified route and length of time -to a
dose of chemical that is likely to be without a measurable risk of adverse, noncancerous effects.
An MRL should not be used as a predictor of adverse health effects. A list of MRLs can be found
at http://www.atsdr.cdc.gov/mrls.html.
Reference Dose (RJD) -Developed by EPA
An RID is an estimate, with safety factors built in, of the daily, life-time exposure of human
populations to a possible hazard that is not likely to cause noncancerous health effects. RfDs can
be found at http://www.epa.gov/iris.
If the estimated exposure dose for a chemical is less than the health guideline value, then the
exposure is unlikely to cause a noncarcinogenic health effect in that specific situation. If the
exposure dose for a chemical is greater than the health guideline, then the exposure dose is
compared to known toxicologic values for that chemical and is discussed in more detail. These
toxicologic values are doses derived from human and animal studies that are summarized in the
ATSDR Toxicological Profiles. A direct comparison of site-specific exposure and doses to
study-derived exposures and doses that cause adverse health effects is the basis for deciding
whether health effects are likely or not.
Calculation of Risk of Carcinogenic Effects
The theoretical risk of developing cancer resulting from exposure to the contaminants was
calculated by multiplying the site-specific adult exposure dose by EPA's corresponding oral slope
factor (found at http://www.epa.gov/iris ). The results estimate the maximum increase in risk of
developing cancer after 70 years of exposure to the contaminant. The risk was then multiplied by
the fraction (25/70) because the exposure was assumed to last 25 years.
The actual risk of cancer is probably lower than the calculated number, which gives a worst-case
excess cancer risk. The method used to calculate EP A's oral slope factor assumes that high-dose
animal data can be used to estimate the risk for low dose exposures in humans. The method also
assumes that no safe level exists for exposure. Little experimental evidence exists to confirm or
refute those two assumptions. Lastly, the method computes the 95% upper bound for the risk,
rather than the average risk, suggesting that the cancer risk is actually lower, perhaps by several
orders of magnitude.2
2 U.S. Environmental Protection Agency (EPA), Office of Emergency and Remedial Response. Risk assessment
guidance for Superfund, volume 1, human health evaluation manual. Washington (DC): U.S. Environmental
Protection Agency; 1989.
3 Agency for Toxic Substances and Disease Registry (ATSDR). Cancer policy framework. Atlanta (GA): U.S.
Department of Health and Human Services; 1993.
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Because of uncertainties involved in estimating carcinogenic risk, A TSDR employs a weight-of-
evidence approach in evaluating all relevant data.3 Therefore, the carcinogenic risk is described
in words (qualitatively) rather than giving a numerical risk estimate only. The numerical risk
estimate must be considered in the context of the variables and assumptions involved in their
derivation and in the broader context of biomedical opinion, host factors, and actual exposure
conditions. The actual parameters of environmental exposures must be given careful
consideration in evaluating the assumptions and variables relating to both toxicity and exposure.
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Public Health Assessment Publie Comment Release Ward Transformer NPL Site
Appendix B. Exposure Pathways for \Vard Transformer Site
Source for all d 0 athwavs: Jperations at \Var Transfonner Site
Pathway Environmental Media & Point of Exposure Route of Exposure Exposure Population Time Notes Name Transport Mechanisms Complete?
'
Site soils and soil at Incidental Population might
Soil Spills from past operations surrounding ingestion, Nearby workers> Past, present; mclude children y
properties inhalation, dennal trespassers (unlikely) future IO years and
exposure older.
Runoff into lagoon; past Water in unnamed lncidental Population might release of contaminated jtributary, Little
jsurface water water to streams; Brier Creek ingestion1 Trespassers at streams Past, present, include children y
dissolution from ;possibly Cr;btree inhalation, dermal dov.11stream from site future 10 years and i
!contaminated sediments Lake exposure older,
!Deposition from surface Population might jwater runoff into lagoon; Lagoon, along Incidental
Sediments streams downstream ingestion, dermal Trespassers at streams Past, presentt include children y ,past release of downstream from site furure 10 years and !contaminated water to of site exposure older, !streams
' JBioaccumulation of
Recreational fishers Past, present1
Population might
Fish ,contaminants from surface 'Meal prepared usini Ingestion include young y
water and sediments into !fish from site area and their families future children fish
Groundwater wells Ingestion; Residents and workers Past, present, IPopulation might
Groundwater Infiltration to groundwater supplying drinking inhalation, dermal
1
near the site future !include young N
water taps exposure 1children
Past release of Inhalation, dermal !Residents and workers jPopu!ation might
Air !contaminants from bumoff Area around site exposure near the site Past !include young N
oven 1children
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Appendix C. ATSDR Glossary of Environmental Health Terms
The Agency for Toxic Substances and Disease Registry (ATSDR) is a federal public health
agency with headquarters in Atlanta, Georgia, and 10 regional offices in the United States.
ATSDR's mission is to serve the public by using the best science, taking responsive public health
actions, and providing trusted health information to prevent harmful exposures and diseases
related to toxic substances. ATSDR is not a regulatory agency, unlike the U.S. Environmental
Protection Agency (EPA), which is the federal agency that develops and enforces environmental
laws to protect the environment and human health.
This glossary defines words used by ATSDR in communications with the public. It is not a
complete dictionary of environmental health terms. Jfyou have questions or comments, call
ATSDR's toll-free telephone number, 1-888-42-ATSDR (1-888-422-8737).
Absorption
Acute Exposure
Additive Effect
Adverse Health
Effect
Antagonistic
Effect
ATSDR
Background Level
Bioavailability
How a chemical enters a person's blood after the chemical has been
swallowed, has come into contact with the skin, or has been breathed in.
Contact with a chemical that happens once or only for a limited period
of time. A TSDR defines acute exposures as those that might last up to
14 days.
A response to a chemical mixture, or combination of substances, that
might be expected if the known effects of individual chemicals, seen al
specific doses, were added together.
A change in body function or the structures of cells that can lead to
disease or health problems.
A response to a mixture of chemicals or combination of substances that
is less than might be expected if the known effects of individual
chemicals, seen at specific doses, were added together.
The Agency for Toxic Substances and Disease Registry. ATSDR is a
federal health agency in Atlanta, Georgia that deals with hazardous
substance and waste site issues. A TSDR gives people information about
harmful chemicals in their environment and tells people how to protect
themselves from coming into contact with chemicals.
An average or expected amount of a chemical in a specific environment.
Or, amounts of chemicals that occur naturally in a specific environment.
See Relative Bioavailability.
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Biota
Cancer
Cancer Slope
Factor (CSF)
Carcinogen
Chronic Exposure
Completed
Exposure
Pathway
Community
Assistance Panel
(CAP)
Comparison
Value (CV)
Comprehensive
Environmental
Response,
Compensation,
and Liability Act
(CERCLA)
Concentration
Contaminant
Delayed Health
Effect
Used in public health, things that humans would eat -including
animals, fish and plants.
A group of diseases which occur when cells in the body become
abnormal and grow, or multiply, out of control
The slope of the dose-response curve for cancer. Multiplying the CSF
by the dose gives a prediction of excess cancer risk for a contaminant.
Any substance shown to cause tumors or cancer in experimental studies.
A contact with a substance or chemical that happens over a long period
of time. A TSDR considers exposures of more than one year to be
chronic.
See Exposure Pathway.
A group of people from the community and health and environmental
agencies who work together on issues and problems at hazardous waste
sites.
Concentrations of substances in air, water, food, and soil that are
unlikely, upon exposure, to cause adverse health effects. Comparison
values are used by health assessors to select which substances and
environmental media (air, water, food and soil) need additional
evaluation while health concerns or effects are investigated.
CERCLA was put into place in 1980. It is also known as Superfund.
This act concerns releases of hazardous substances into the
environment, and the cleanup of these substances and hazardous waste
sites. This act created ATSDR and gave it the responsibility to look into
health issues related to hazardous waste sites.
How much or the amount of a substance present in a certain amount of
soil, water, air, or food.
See Environmental Contaminant.
A disease or injury that happens as a result of exposures that may have
occurred far in the past.
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Dermal Contact
Dose
Dose/ Response
Duration
Environmental
Contaminant
Environmental
Media
U.S.
Environmental
l'rotcction Agency
(EPA)
Epidemiology
Exposure
Exposure
Assessment
A chemical getting onto your skin (see Route or Exposure).
The amount of a substance to which a person may be exposed, usually
on a daily basis. Dose is often explained as "amount of substance(s) per
body weight per day".
The relationship between the amount of exposure (dose) and the change
in body function or health that results.
The amount of time (days, months, years) that a person is exposed to a
chemical.
A substance ( chemical) that gets into a system (person, animal, or the
environment) in amounts higher than the Background Level, or what
would be expected.
Usually refers to the air, water, and soil in which chemicals of interest
are found. Sometimes refers to the plants and animals that are eaten by
humans. Environmental Media is the second part of an Exposure
Pathway.
The federal agency that develops and enforces environmental laws to
protect the environment and the public's health.
The study of the different factors that determine how often, in how
many people, and in which people will disease occur.
Coming into contact with a chemical substance. (For the three ways
people can come in contact with substances, see Route of Exposure.)
The process of finding the ways people come in contact with chemicals,
how often and how long they come in contact with chemicals, and the
amounts of chemicals with which they come in contact.
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Exposure
Pathway
Frequency
Hazardous Waste
Health Effect
Indeterminate
Public Health
Hazard
Ingestion
Inhalation
LOAEL
Malignancy
A description of the way that a chemical moves from its source (where
it began) to where and how people can come into contact with (or get
exposed to) the chemical.
ATSDR defines an exposure pathway as having 5 parts:
1. Source of Contamination,
2. Environmental Media and Transport Mechanism,
3. Point of Exposure,
4. Route of Exposure, and
5. Receptor Population.
When all 5 parts of an exposure pathway are present, it is called a
Completed Exposure Pathway. Each of these 5 terms is defined in this
Glossary.
How often a person is exposed to a chemical over time; for example,
every day, once a week, twice a month.
Substances that have been released or thrown away into the
environment and, under certain conditions, could be harmful to people
who come into contact with them.
ATSDR deals only with Adverse Health Effects (see definition in this
Glossary).
The category is used in Public Health Assessment documents for sites
where important information is lacking (missing or has not yet been
gathered) about site-related chemical exposures.
Swallowing something, as in eating or drinking. It is a way a chemical
can enter your body (see Route of Exposure).
Breathing. It is a way a chemical can enter your body (see Route of
Exposure).
Lowest Observed Adverse Effect Level. The lowest dose of a chemical
in a study, or group of studies, that has caused harmful health effects in
people or animals.
See Cancer.
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MRL
NPL
NOAEL
No Apparent
Public Health
Hazard
No Public Health
Hazard
PHA
Plume
Point of Exposure
Population
Minimal Risk Level. An estimate of daily human exposure -by a
specified route and length of time•-to a dose of chemical that is likely
to be without a measurable risk of adverse, noncancerous effects. An
MRL should not be used as a predictor of adverse health effects.
The National Priorities List. (Which is part ofSuperfund.) A list kept
by the U.S. Enviromnental Protection Agency (EPA) of the most serious
uncontrolled or abandoned hazardous waste sites in tbe country. An
NPL site needs to be cleaned up or is being looked at to see if people
can be exposed to chemicals from the site.
No Observed Adverse Effect Level. The highest dose of a chemical in a
study, or group of studies, that did nol cause harmful health effects in
people or animals.
The category is used in ATSDR's Public Health Assessment documents
for sites where exposure to site-related chemicals may have occurred in
the past or is still occurring but the exposures are nol at levels expected
to cause adverse health effects.
The category is used in A TSDR's Public Health Assessment documents
for sites where there is evidence of an absence of exposure to site-
related chemicals.
Public Health Assessment. A report or document that looks at chemicals
at a hazardous waste site and tells if people could be harmed from
coming into contact with those chemicals. The PHA also tells if
possible further public health actions are needed.
A line or column of air or water containing chemicals moving from the
source to areas further away. A plume can be a column or clouds of
smoke from a chimney or contaminated underground water sources or
contaminated surface water (such as lakes, ponds and streams).
The place where someone can come into contact with a contaminated
environmental medium (air, water, food or soil). Some exalllp!es
include: the area of a playground that has contaminated dirt, a
contaminated spring used for drinking water, or the backyard area where
someone might breathe contaminated air.
A group of people living in a certain area; or the number of people in a
certain area.
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Public Health Assessment --Public Comment Release Ward Transformer NPL Site
PRP
Public Health
Asscssment(s)
Public Health
Hazard
Public Health
Hazard Criteria
Receptor
Population
Reference Dose
(RID)
Relative
Bioavailability
Route of Exposure
Safety Factor
Potentially Responsible Party. A company, government or person that is
responsible for causing the pollution at a hazardous waste site. PRP's
are expected to help.pay for the clean up of a site.
See PHA.
The category is used in PH As for sites that have certain physical
features or evidence of chronic, site-related chemical exposure that
could result in adverse health effects.
PHA categories given to a site which tel! whether people could be
harmed by conditions present at the site. Each are defined in the
Glossary. The categories are:
-Urgent Public Health Hazard
-Public Health Hazard
-Indeterminate Public Health Hazard
-No Apparent Public Health Hazard
-No Public Health Hazard
People who live or work in the path of one or more chemicals, and who
could come into contact with them (Sec Exposure Pathway).
An estimate, with safety factors (see safety factor) built in, of the daily,
life-time exposure of human populations to a possible hazard that is not
likely to cause harm to the person.
The amount of a compound that can be absorbed from a particular
medium (such as soil) compared to the amount absorbed from a
reference material (such as water). Expressed in percentage fonn.
The way a chemical can get into a person's body. There are three
exposure routes:
-breathing (also called inhalation),
-eating or drinking (also called ingestion), and
getting something on the skin ( also called dermal contact).
Also called Uncertainty Factor. When scientists don't have enough
information to decide if an exposure will cause harm to people, they use
"safety factors" and formulas in place of the information that is not
known. These factors and formulas can help determine the amount of a
chemical that is not likely to cause harm to people.
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SARA
Sample Size
Sample
Source
(of
Contamination)
Special
Populations
Statistics
Superfund Site
Survey
The Superfund Amendments and Reauthorization Act in 1986 amended
CERCLA (see CERCLA) and expanded the health-related
responsibilities of A TSDR. CERCLA and SARA direct ATSDR lo look
into the health effects resulting from chemical exposures al hazardous
waste sites.
The number of people that are needed for a health study.
A small number of people chosen from a larger population (see
Population).
The place where a chemical comes from, such as a landfill, pond, creek,
incinerator, tank, or drnm. Contaminant source is the first part of an
Exposure Pathway.
People who may be more sensitive to chemical exposures because of
certain factors such as age, a disease they already have, occupation, sex,
or certain behaviors (like cigarette smoking). Children, pregnant
women, and older people are often considered special populations.
A branch of the math process of collecting, looking at, and summarizing
data or infonnation.
See NPL.
A way to collect information or data from a group of people
(population). Surveys can be done by phone, mail, or in person.
ATSDR cannot do surveys of more than nine people without approval
from the U.S. Department of Health and Human Services.
Synergistic Effect A health effect from an exposure to more than one chemical, where one
of the chemicals worsens the effect of another chemical. The combined
effect of the chemicals acting together are greater than the effects of the
chemicals acting by themselves,
Toxic
Toxicology
Tumor
Harmful. Any substance or chemical can be toxic at a certain dose
(amount). The dose is what detem1ines the potential harm of a chemical
and whether it would cause someone to get sick.
The study of the harmful effects of chemicals on humans or animals.
Abnormal growth of tissue or cells that have formed a lump or mass.
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Public Health Assessment -Public Comment Release Ward Transformer NPL Site
Uncertainty
Factor
Urgent Public
Health Hazard
See Safety Factor.
This category is used in ATSDR's Public Health Assessment documents
for sites that have certain physical features or evidence of short-term
(less than 1 year), site-related chemical exposure that could result in
adverse health effects and require quick intervention to stop people from
being exposed.
36